About the journal
JAK-STAT is a journal published by Taylor & Francis in United Kingdom. It is listed in PUBMED according to the sources read. Facts are reproduced as recorded at their sources with their capture dates; PublishLens indicators are available after sign-in.
Basic facts
- Publisher
- Taylor & Francis
- ISSN
- 2162-3988 · 2162-3996
- Country
- United Kingdom
- Access
- Not open access / not declared
- Declared peer-review model
- peer_review
- Official website
- http://www.tandfonline.com/toc/kjks20/current#.VdyHxv6FPcs
Index listings
- PUBMED · 2012-01-01
- Indexing
- PUBMED
- APC
- —
- Declared review time (DOAJ)
- —
- Activity (OpenAlex)
- Dormant — no works recorded since 2016
- PublishLens list status
- Shown after sign-in
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Methodology signals (dated facts from their sources)
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Descriptive metadata from public sources (DOAJ, OpenAlex, PubMed) as of the capture date shown — contains no assessment or judgment.
Works in OpenAlex
151
Last recorded publication year
2016
h-index
50
i10-index
123
From OpenAlex open data (CC0).
Editorial & policy3
- Observed digital preservation (Keepers registry)— snapshot 2026-10-05
- CLOCKSSPortico
- Open references deposited at Crossref
- 0%
- ORCID iDs in current deposits
- 0%
Identity & continuity3
- Alternate titles
- JAKSTAT
- Source type
- journal
- NLM unique ID (PubMed)
- 101591376
Classification(no data)
No data available to the platform for this group
No platform data for: Classification — absence is not a judgment about the journal.
Subject areas
- Cytokine Signaling Pathways and Interactions
- Cytokine Signaling Pathways and Interactions
- Cytokine Signaling Pathways and Interactions
- interferon and immune responses
- interferon and immune responses
- Immune Cell Function and Interaction
- NF-κB Signaling Pathways
- Immune Cell Function and Interaction
Show all subjects (75)
- Protein Tyrosine Phosphatases
- interferon and immune responses
- Protein Tyrosine Phosphatases
- Protein Tyrosine Phosphatases
- Developmental Biology and Gene Regulation
- TGF-β signaling in diseases
- T-cell and B-cell Immunology
- NF-κB Signaling Pathways
- NF-κB Signaling Pathways
- Receptor Mechanisms and Signaling
- Cancer Mechanisms and Therapy
- Adipokines, Inflammation, and Metabolic Diseases
- T-cell and B-cell Immunology
- Myeloproliferative Neoplasms: Diagnosis and Treatment
- Myeloproliferative Neoplasms: Diagnosis and Treatment
- PI3K/AKT/mTOR signaling in cancer
- Genomics and Chromatin Dynamics
- Cancer Mechanisms and Therapy
- T-cell and B-cell Immunology
- PI3K/AKT/mTOR signaling in cancer
- Cardiac Ischemia and Reperfusion
- Cardiac Fibrosis and Remodeling
- Myeloproliferative Neoplasms: Diagnosis and Treatment
- IL-33, ST2, and ILC Pathways
- Invertebrate Immune Response Mechanisms
- Invertebrate Immune Response Mechanisms
- IL-33, ST2, and ILC Pathways
- Estrogen and related hormone effects
- RNA Research and Splicing
- Reproductive System and Pregnancy
- Adipokines, Inflammation, and Metabolic Diseases
- RNA Research and Splicing
- Reproductive System and Pregnancy
- Psoriasis: Treatment and Pathogenesis
- Adipokines, Inflammation, and Metabolic Diseases
- MicroRNA in disease regulation
- Immunodeficiency and Autoimmune Disorders
- Nuclear Receptors and Signaling
- MicroRNA in disease regulation
- Nuclear Receptors and Signaling
- Immunodeficiency and Autoimmune Disorders
- Immune Response and Inflammation
- Growth Hormone and Insulin-like Growth Factors
- Eosinophilic Disorders and Syndromes
- Acute Myeloid Leukemia Research
- Eosinophilic Disorders and Syndromes
- Galectins and Cancer Biology
- CRISPR and Genetic Engineering
- Immune Response and Inflammation
- Digestive system and related health
- Chronic Myeloid Leukemia Treatments
- Growth Hormone and Insulin-like Growth Factors
- CRISPR and Genetic Engineering
- Kruppel-like factors research
- Mechanisms of cancer metastasis
- Digestive system and related health
- Immune Response and Inflammation
- Kruppel-like factors research
- Galectins and Cancer Biology
- Pluripotent Stem Cells Research
- Growth Hormone and Insulin-like Growth Factors
- Chronic Myeloid Leukemia Treatments
- MicroRNA in disease regulation
- Regulation of Appetite and Obesity
- Hematopoietic Stem Cell Transplantation
- Cancer Cells and Metastasis
- Protein Kinase Regulation and GTPase Signaling
Community confirmations — what users saw in MJL or Scopus Sources; not platform verification and never part of any score.
The journal's record in other sources:Web of Science MJLDOAJ