About the journal
Acta Pharmacologica Sinica is a journal published by Springer Nature in United Kingdom. It is listed in MEDLINE according to the sources read. Declared publication charge: 3,990 EUR. Facts are reproduced as recorded at their sources with their capture dates; PublishLens indicators are available after sign-in.
Basic facts
- Publisher
- Springer Nature
- ISSN
- 1671-4083 · 1745-7254
- Country
- United Kingdom
- Access
- Not open access / not declared
- Declared publication charge
- 3,990 EUR
- Declared peer-review model
- peer_review
- Official website
- https://www.nature.com/aps/
Index listings
- MEDLINE · 2000-01-01
- Indexing
- MEDLINE
- APC
- 3,990 EUR
- Declared review time (DOAJ)
- —
- Actual review time (measured)
- 140 days received→accepted — computed from PubMed (n=95)
- Activity (OpenAlex)
- Active — latest works 2026
- PublishLens list status
- Shown after sign-in
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Methodology signals (dated facts from their sources)
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Descriptive metadata from public sources (DOAJ, OpenAlex, PubMed) as of the capture date shown — contains no assessment or judgment.
Works in OpenAlex
4553
Last recorded publication year
2026
h-index
145
i10-index
3389
From OpenAlex open data (CC0).
Editorial & policy5
- Observed digital preservation (Keepers registry)— snapshot 2026-10-05
- CLOCKSSPortico
- Norwegian register (HK-dir)— 2026
- Level 1 — approved scholarly channel
- Open references deposited at Crossref
- 95%
- ORCID iDs in current deposits
- 3%
- Enrolled in Crossref Similarity Check
- ✓
- Deposits licence metadata at Crossref
- ✓
Identity & continuity4
- Alternate titles
- Zhongguo yaoli xuebaoAPS
- Source type
- journal
- NLM unique ID (PubMed)
- 100956087
- Society affiliations
- Shanghai Institute of Materia Medica (SIMM), Chinese Academy of Sciences
Classification(no data)
No data available to the platform for this group
No platform data for: Classification — absence is not a judgment about the journal.
Subject areas
- Autophagy in Disease and Therapy
- Autophagy in Disease and Therapy
- Neuroscience and Neuropharmacology Research
- Neuroscience and Neuropharmacology Research
- Ion channel regulation and function
- Ion channel regulation and function
- Receptor Mechanisms and Signaling
- Receptor Mechanisms and Signaling
Show all subjects (50)
- Neuroinflammation and Neurodegeneration Mechanisms
- Neuroinflammation and Neurodegeneration Mechanisms
- Traditional Chinese Medicine Analysis
- Traditional Chinese Medicine Analysis
- Cell death mechanisms and regulation
- Cell death mechanisms and regulation
- Cardiac electrophysiology and arrhythmias
- Cardiac electrophysiology and arrhythmias
- Cancer, Hypoxia, and Metabolism
- Drug Transport and Resistance Mechanisms
- Drug Transport and Resistance Mechanisms
- Ubiquitin and proteasome pathways
- Cancer, Hypoxia, and Metabolism
- Ubiquitin and proteasome pathways
- Neuropeptides and Animal Physiology
- Neuropeptides and Animal Physiology
- Neurotransmitter Receptor Influence on Behavior
- Neurotransmitter Receptor Influence on Behavior
- Liver Disease Diagnosis and Treatment
- Liver Disease Diagnosis and Treatment
- Immune Response and Inflammation
- Immune Response and Inflammation
- Nitric Oxide and Endothelin Effects
- Nicotinic Acetylcholine Receptors Study
- Nitric Oxide and Endothelin Effects
- Cardiac Ischemia and Reperfusion
- Cardiac Ischemia and Reperfusion
- Nicotinic Acetylcholine Receptors Study
- Endoplasmic Reticulum Stress and Disease
- Endoplasmic Reticulum Stress and Disease
- MicroRNA in disease regulation
- Inflammasome and immune disorders
- Inflammasome and immune disorders
- MicroRNA in disease regulation
- Pharmacogenetics and Drug Metabolism
- Pharmacogenetics and Drug Metabolism
- Computational Drug Discovery Methods
- Cardiac Fibrosis and Remodeling
- Protein Kinase Regulation and GTPase Signaling
- Computational Drug Discovery Methods
- Cardiac Fibrosis and Remodeling
- Protein Kinase Regulation and GTPase Signaling
Community confirmations — what users saw in MJL or Scopus Sources; not platform verification and never part of any score.
The journal's record in other sources:Web of Science MJLDOAJ