Small GTPases

Taylor & Francis

Journal websitePeer review: Peer review
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About the journal

Small GTPases is a journal published by Taylor & Francis in United States. It is listed in PUBMED according to the sources read. Facts are reproduced as recorded at their sources with their capture dates; PublishLens indicators are available after sign-in.

Basic facts

Publisher
Taylor & Francis
ISSN
2154-1248 · 2154-1256
Country
United States
Access
Not open access / not declared
Declared peer-review model
peer_review

Index listings

  • PUBMED · 2011-11-01
Indexing
PUBMED
APC
—
Declared review time (DOAJ)
—
Activity (OpenAlex)
Active — latest works 2024
PublishLens list status
Shown after sign-in
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Methodology signals (dated facts from their sources)

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Journal profile

Descriptive metadata from public sources (DOAJ, OpenAlex, PubMed) as of the capture date shown — contains no assessment or judgment.

Works in OpenAlex

552

Last recorded publication year

2024

Reported by OpenAlex in 1 of 3 sources (CC0 flags, not verified indexing)DOAJSciELOOpenAlex Core

h-index

67

i10-index

402

From OpenAlex open data (CC0).

Editorial & policy4
Observed digital preservation (Keepers registry)— snapshot 2026-10-05
CLOCKSSPortico
Open references deposited at Crossref
100%
ORCID iDs in current deposits
67%
Enrolled in Crossref Similarity Check
✓
Deposits licence metadata at Crossref
✓
Identity & continuity3
Alternate titles
Small Gtpases
Source type
journal
NLM unique ID (PubMed)
101530974
Classification(no data)

No data available to the platform for this group

No platform data for: Classification — absence is not a judgment about the journal.

Subject areas

  • Cellular transport and secretion
  • Cellular transport and secretion
  • Protein Kinase Regulation and GTPase Signaling
  • Protein Kinase Regulation and GTPase Signaling
  • Cellular Mechanics and Interactions
  • Cellular Mechanics and Interactions
  • Microtubule and mitosis dynamics
  • Microtubule and mitosis dynamics
Show all subjects (50)
  • Cell Adhesion Molecules Research
  • Cell Adhesion Molecules Research
  • Hippo pathway signaling and YAP/TAZ
  • Hippo pathway signaling and YAP/TAZ
  • PI3K/AKT/mTOR signaling in cancer
  • PI3K/AKT/mTOR signaling in cancer
  • Retinal Development and Disorders
  • Retinal Development and Disorders
  • Wnt/β-catenin signaling in development and cancer
  • Wnt/β-catenin signaling in development and cancer
  • Cancer-related Molecular Pathways
  • Cancer-related Molecular Pathways
  • Endoplasmic Reticulum Stress and Disease
  • Cell death mechanisms and regulation
  • Endoplasmic Reticulum Stress and Disease
  • Cell death mechanisms and regulation
  • Erythrocyte Function and Pathophysiology
  • Erythrocyte Function and Pathophysiology
  • Ubiquitin and proteasome pathways
  • Melanoma and MAPK Pathways
  • Ubiquitin and proteasome pathways
  • Melanoma and MAPK Pathways
  • Pancreatic function and diabetes
  • Pancreatic function and diabetes
  • Lipid Membrane Structure and Behavior
  • Lipid Membrane Structure and Behavior
  • Fungal and yeast genetics research
  • Fungal and yeast genetics research
  • Genetics, Aging, and Longevity in Model Organisms
  • Calcium signaling and nucleotide metabolism
  • Calcium signaling and nucleotide metabolism
  • Autophagy in Disease and Therapy
  • Autophagy in Disease and Therapy
  • Genetics, Aging, and Longevity in Model Organisms
  • Metabolism, Diabetes, and Cancer
  • Receptor Mechanisms and Signaling
  • Metabolism, Diabetes, and Cancer
  • Receptor Mechanisms and Signaling
  • Mitochondrial Function and Pathology
  • RNA Research and Splicing
  • RNA Research and Splicing
  • Mitochondrial Function and Pathology

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The journal's record in other sources:Web of Science MJLDOAJ

Everything shown here is produced automatically from named sources (DOAJ, OpenAlex, Crossref, PubMed, the Norwegian register) as of its capture date, with no human editing. Facts appear as recorded at the source; derived scores (PCI, PTD, PublishLens score) follow a published methodology. Not a judgement on the journal, not an official accreditation, not advice.

Wrong items are corrected automatically by re-reading the source (24 h for daily sources, 7 days for weekly ones) with a dated change log, and each item links to its source record. PublishLens-derived assessments are contested through the appeals mechanism (15 working days). Appeals · Indicator guide

Norwegian register data: Kanalregisteret (HK-dir, Norway) — CC BY 4.0 / NLOD — adapted: level extracted per year · OASPA member list — CC BY

Last automated source read: