Cellular Signalling

Elsevier BV

Journal websitePeer review: Peer review
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About the journal

Cellular Signalling is a journal published by Elsevier BV in United Kingdom. It is listed in MEDLINE and PUBMED according to the sources read. Declared publication charge: 3,010 USD. Facts are reproduced as recorded at their sources with their capture dates; PublishLens indicators are available after sign-in.

Basic facts

Publisher
Elsevier BV
ISSN
1873-3913 · 0898-6568
Country
United Kingdom
Access
Not open access / not declared
Declared publication charge
3,010 USD
Declared peer-review model
peer_review

Index listings

  • MEDLINE · 1989-01-01
  • PUBMED · 2008-07-11
Indexing
MEDLINE · PUBMED
APC
3,010 USD
Declared review time (DOAJ)
—
Actual review time (measured)
87 days received→accepted — computed from PubMed (n=90)
Activity (OpenAlex)
Active — latest works 2026
PublishLens list status
Shown after sign-in
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Methodology signals (dated facts from their sources)

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Journal profile

Descriptive metadata from public sources (DOAJ, OpenAlex, PubMed) as of the capture date shown — contains no assessment or judgment.

Works in OpenAlex

8095

Last recorded publication year

2026

Reported by OpenAlex in 1 of 3 sources (CC0 flags, not verified indexing)DOAJSciELOOpenAlex Core

h-index

192

i10-index

5273

From OpenAlex open data (CC0).

Editorial & policy4
Observed digital preservation (Keepers registry)— snapshot 2026-10-05
CLOCKSSPortico
Norwegian register (HK-dir)— 2026
Level 1 — approved scholarly channel
Open references deposited at Crossref
94%
ORCID iDs in current deposits
1%
Deposits licence metadata at Crossref
✓
Identity & continuity2
Source type
journal
NLM unique ID (PubMed)
8904683
Classification(no data)

No data available to the platform for this group

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Subject areas

  • Protein Kinase Regulation and GTPase Signaling
  • Protein Kinase Regulation and GTPase Signaling
  • Receptor Mechanisms and Signaling
  • Receptor Mechanisms and Signaling
  • Cancer-related molecular mechanisms research
  • Cancer-related molecular mechanisms research
  • PI3K/AKT/mTOR signaling in cancer
  • PI3K/AKT/mTOR signaling in cancer
Show all subjects (50)
  • Cell Adhesion Molecules Research
  • Cell Adhesion Molecules Research
  • Ubiquitin and proteasome pathways
  • Ubiquitin and proteasome pathways
  • Diverse Scientific and Economic Studies
  • Diverse Scientific and Economic Studies
  • Human auditory perception and evaluation
  • Human auditory perception and evaluation
  • Cellular transport and secretion
  • Cellular transport and secretion
  • NF-κB Signaling Pathways
  • NF-κB Signaling Pathways
  • Cytokine Signaling Pathways and Interactions
  • Cytokine Signaling Pathways and Interactions
  • MicroRNA in disease regulation
  • MicroRNA in disease regulation
  • RNA modifications and cancer
  • RNA modifications and cancer
  • Ion channel regulation and function
  • Ion channel regulation and function
  • Autophagy in Disease and Therapy
  • Cell death mechanisms and regulation
  • Cell death mechanisms and regulation
  • Autophagy in Disease and Therapy
  • Educational Robotics and Engineering
  • Educational Robotics and Engineering
  • Metabolism, Diabetes, and Cancer
  • Metabolism, Diabetes, and Cancer
  • Endoplasmic Reticulum Stress and Disease
  • Endoplasmic Reticulum Stress and Disease
  • Ferroptosis and cancer prognosis
  • Neuropeptides and Animal Physiology
  • Neuropeptides and Animal Physiology
  • Ferroptosis and cancer prognosis
  • Cancer-related Molecular Pathways
  • Circular RNAs in diseases
  • Circular RNAs in diseases
  • Wnt/β-catenin signaling in development and cancer
  • Cancer-related Molecular Pathways
  • Wnt/β-catenin signaling in development and cancer
  • interferon and immune responses
  • Pancreatic function and diabetes

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The journal's record in other sources:Web of Science MJLDOAJ

Everything shown here is produced automatically from named sources (DOAJ, OpenAlex, Crossref, PubMed, the Norwegian register) as of its capture date, with no human editing. Facts appear as recorded at the source; derived scores (PCI, PTD, PublishLens score) follow a published methodology. Not a judgement on the journal, not an official accreditation, not advice.

Wrong items are corrected automatically by re-reading the source (24 h for daily sources, 7 days for weekly ones) with a dated change log, and each item links to its source record. PublishLens-derived assessments are contested through the appeals mechanism (15 working days). Appeals · Indicator guide

Norwegian register data: Kanalregisteret (HK-dir, Norway) — CC BY 4.0 / NLOD — adapted: level extracted per year · OASPA member list — CC BY

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