Cell Death Discovery

Nature Publishing Group

Journal websitePeer review: Peer review
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About the journal

Cell Death Discovery is an open-access journal published by Nature Publishing Group in United Kingdom, publishing in english. It is listed in DOAJ and PUBMED according to the sources read. Declared publication charge: 2,490 EUR. Declared review time at DOAJ: 9 weeks. Facts are reproduced as recorded at their sources with their capture dates; PublishLens indicators are available after sign-in.

Basic facts

Publisher
Nature Publishing Group
ISSN
2058-7716
Country
United Kingdom
Languages
english
Access
Open access
Declared publication charge
2,490 EUR
Articles in DOAJ
2809
Declared review time
9 weeks
Declared peer-review model
peer_review

Index listings

  • DOAJ · 2018-09-10
  • PUBMED · 2015-08-17
Indexing
DOAJ · PUBMED
APC
2,490 EUR
Declared review time (DOAJ)
9 weeks — journal-declared only (not measured)
Activity (OpenAlex)
Active — latest works 2026
PublishLens list status
Shown after sign-in
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Methodology signals (dated facts from their sources)

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Journal profile

Descriptive metadata from public sources (DOAJ, OpenAlex, PubMed) as of the capture date shown — contains no assessment or judgment.

Articles in DOAJ

2809

Most recent article

last month

Average review time (journal-declared via DOAJ)

9weeks

Works in OpenAlex

3458

Last recorded publication year

2026

Reported by OpenAlex in 2 of 3 sources (CC0 flags, not verified indexing)DOAJSciELOOpenAlex Core

h-index

103

i10-index

2053

From OpenAlex open data (CC0).

Editorial & policy13
Last DOAJ review date
2026-03-17
Added to DOAJ
2018-09-10
Declared plagiarism screening
Declared
Other fees beyond APC
No
Declared digital preservation
PMCPortico
Observed digital preservation (Keepers registry)— snapshot 2026-10-05
CLOCKSSPortico
Editorial board
Official page ↗
Norwegian register (HK-dir)— 2026
Level 1 — approved scholarly channel
Open references deposited at Crossref
91%
ORCID iDs in current deposits
98%
Enrolled in Crossref Similarity Check
✓
Deposits licence metadata at Crossref
✓
APC waiver policy
View policy
Peer-review policy
View policy
Identity & continuity2
Source type
journal
NLM unique ID (PubMed)
101665035
Classification4

Subjects (DOAJ)

Medicine: Internal medicine: Neoplasms. Tumors. Oncology. Including cancer and carcinogensScience: Biology (General): Cytology

LCC codes

RC254-282QH573-671

Subject areas

  • RNA modifications and cancer
  • RNA modifications and cancer
  • Cancer-related molecular mechanisms research
  • Cancer-related molecular mechanisms research
  • Ferroptosis and cancer prognosis
  • Ferroptosis and cancer prognosis
  • Autophagy in Disease and Therapy
  • Autophagy in Disease and Therapy
Show all subjects (50)
  • MicroRNA in disease regulation
  • MicroRNA in disease regulation
  • Cancer, Hypoxia, and Metabolism
  • Cancer, Hypoxia, and Metabolism
  • Epigenetics and DNA Methylation
  • Epigenetics and DNA Methylation
  • Circular RNAs in diseases
  • Circular RNAs in diseases
  • Ubiquitin and proteasome pathways
  • Ubiquitin and proteasome pathways
  • Cell death mechanisms and regulation
  • Cell death mechanisms and regulation
  • RNA Research and Splicing
  • Inflammasome and immune disorders
  • RNA Research and Splicing
  • Inflammasome and immune disorders
  • Endoplasmic Reticulum Stress and Disease
  • Endoplasmic Reticulum Stress and Disease
  • Immune cells in cancer
  • Immune cells in cancer
  • Cancer-related Molecular Pathways
  • Cancer-related Molecular Pathways
  • Cancer-related gene regulation
  • Cancer-related gene regulation
  • Mitochondrial Function and Pathology
  • Mitochondrial Function and Pathology
  • Cancer, Lipids, and Metabolism
  • Cancer, Lipids, and Metabolism
  • interferon and immune responses
  • Extracellular vesicles in disease
  • Extracellular vesicles in disease
  • interferon and immune responses
  • Cancer Cells and Metastasis
  • Cancer Cells and Metastasis
  • Phagocytosis and Immune Regulation
  • Phagocytosis and Immune Regulation
  • Hippo pathway signaling and YAP/TAZ
  • Hippo pathway signaling and YAP/TAZ
  • DNA Repair Mechanisms
  • Histone Deacetylase Inhibitors Research
  • DNA Repair Mechanisms
  • Histone Deacetylase Inhibitors Research

Keywords (DOAJ)

  • cell death
  • cell biology
  • cancer
  • neurobiology
  • inflammation

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The journal's record in other sources:Web of Science MJLDOAJ

Everything shown here is produced automatically from named sources (DOAJ, OpenAlex, Crossref, PubMed, the Norwegian register) as of its capture date, with no human editing. Facts appear as recorded at the source; derived scores (PCI, PTD, PublishLens score) follow a published methodology. Not a judgement on the journal, not an official accreditation, not advice.

Wrong items are corrected automatically by re-reading the source (24 h for daily sources, 7 days for weekly ones) with a dated change log, and each item links to its source record. PublishLens-derived assessments are contested through the appeals mechanism (15 working days). Appeals · Indicator guide

Norwegian register data: Kanalregisteret (HK-dir, Norway) — CC BY 4.0 / NLOD — adapted: level extracted per year · OASPA member list — CC BY

Last automated source read: