About the journal
Oncogenesis is an open-access journal published by Nature Publishing Group in United Kingdom, publishing in english. It is listed in DOAJ and PUBMED according to the sources read. Declared publication charge: 3,590 EUR. Declared review time at DOAJ: 26 weeks. Facts are reproduced as recorded at their sources with their capture dates; PublishLens indicators are available after sign-in.
Basic facts
- Publisher
- Nature Publishing Group
- ISSN
- 2157-9024
- Country
- United Kingdom
- Languages
- english
- Access
- Open access
- Declared publication charge
- 3,590 EUR
- Articles in DOAJ
- 355
- Declared review time
- 26 weeks
- Declared peer-review model
- peer_review
- Official website
- https://www.nature.com/oncsis/index.html
Index listings
- DOAJ · 2013-05-01
- PUBMED · 2012-02-20
- Indexing
- DOAJ · PUBMED
- APC
- 3,590 EUR
- Declared review time (DOAJ)
- 26 weeks — journal-declared only (not measured)
- Activity (OpenAlex)
- Active — latest works 2026
- PublishLens list status
- Shown after sign-in
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- Choose your university and rank below ↓
Methodology signals (dated facts from their sources)
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Descriptive metadata from public sources (DOAJ, OpenAlex, PubMed) as of the capture date shown — contains no assessment or judgment.
Articles in DOAJ
355
Most recent article
2 months ago
Average review time (journal-declared via DOAJ)
26weeks
Works in OpenAlex
1014
Last recorded publication year
2026
h-index
93
i10-index
799
From OpenAlex open data (CC0).
Editorial & policy13
- Last DOAJ review date
- 2026-03-17
- Added to DOAJ
- 2013-05-01
- Declared plagiarism screening
- Declared
- Other fees beyond APC
- No
- Declared digital preservation
- CLOCKSS
- Observed digital preservation (Keepers registry)— snapshot 2026-10-05
- CLOCKSSPortico
- Editorial board
- Official page ↗
- Norwegian register (HK-dir)— 2026
- Level 1 — approved scholarly channel
- Open references deposited at Crossref
- 85%
- ORCID iDs in current deposits
- 97%
- Enrolled in Crossref Similarity Check
- ✓
- Deposits licence metadata at Crossref
- ✓
- APC waiver policy
- View policy
- Peer-review policy
- View policy
Identity & continuity3
- Alternate titles
- Oncogenesis
- Source type
- journal
- NLM unique ID (PubMed)
- 101580004
Classification2
Subjects (DOAJ)
LCC codes
Subject areas
- Cancer, Hypoxia, and Metabolism
- Cancer, Hypoxia, and Metabolism
- Epigenetics and DNA Methylation
- Epigenetics and DNA Methylation
- RNA modifications and cancer
- RNA modifications and cancer
- Ubiquitin and proteasome pathways
- Ubiquitin and proteasome pathways
Show all subjects (50)
- Cancer Cells and Metastasis
- Cancer Cells and Metastasis
- Cancer-related Molecular Pathways
- Cancer-related Molecular Pathways
- Cancer-related molecular mechanisms research
- Cancer-related molecular mechanisms research
- Cancer-related gene regulation
- Cancer-related gene regulation
- MicroRNA in disease regulation
- MicroRNA in disease regulation
- Wnt/β-catenin signaling in development and cancer
- Wnt/β-catenin signaling in development and cancer
- RNA Research and Splicing
- RNA Research and Splicing
- Circular RNAs in diseases
- Circular RNAs in diseases
- Cell Adhesion Molecules Research
- Cell Adhesion Molecules Research
- DNA Repair Mechanisms
- Histone Deacetylase Inhibitors Research
- DNA Repair Mechanisms
- Histone Deacetylase Inhibitors Research
- PI3K/AKT/mTOR signaling in cancer
- PI3K/AKT/mTOR signaling in cancer
- Immune cells in cancer
- Pancreatic and Hepatic Oncology Research
- Cancer Genomics and Diagnostics
- Glycosylation and Glycoproteins Research
- Cancer Genomics and Diagnostics
- Pancreatic and Hepatic Oncology Research
- Glycosylation and Glycoproteins Research
- Immune cells in cancer
- Hippo pathway signaling and YAP/TAZ
- Peptidase Inhibition and Analysis
- Hippo pathway signaling and YAP/TAZ
- Peptidase Inhibition and Analysis
- Cancer Mechanisms and Therapy
- Cancer Mechanisms and Therapy
- Protein Degradation and Inhibitors
- Protein Degradation and Inhibitors
- Ferroptosis and cancer prognosis
- Ferroptosis and cancer prognosis
Keywords (DOAJ)
- apoptosis
- cancer metabolism
- cellular oncogenes
- dna
- damage and repair
- tumour suppression
Community confirmations — what users saw in MJL or Scopus Sources; not platform verification and never part of any score.
The journal's record in other sources:Web of Science MJLDOAJ