Frontiers in Cell Death

Frontiers Media S.A.

Journal websitePeer review: Peer review
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About the journal

Frontiers in Cell Death is an open-access journal published by Frontiers Media S.A. in Switzerland, publishing in english. It is listed in DOAJ according to the sources read. Declared publication charge: 1,950 CHF. Declared review time at DOAJ: 12 weeks. Facts are reproduced as recorded at their sources with their capture dates; PublishLens indicators are available after sign-in.

Basic facts

Publisher
Frontiers Media S.A.
ISSN
2813-5563
Country
Switzerland
Languages
english
Access
Open access
Declared publication charge
1,950 CHF
Articles in DOAJ
31
Declared review time
12 weeks
Declared peer-review model
peer_review

Index listings

  • DOAJ · 2024-05-17
Indexing
DOAJ
APC
1,950 CHF
Declared review time (DOAJ)
12 weeks — journal-declared only (not measured)
Activity (OpenAlex)
Active — latest works 2026
PublishLens list status
Shown after sign-in
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Methodology signals (dated facts from their sources)

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Journal profile

Descriptive metadata from public sources (DOAJ, OpenAlex, PubMed) as of the capture date shown — contains no assessment or judgment.

Articles in DOAJ

31

Most recent article

last month

Average review time (journal-declared via DOAJ)

12weeks

Works in OpenAlex

40

Last recorded publication year

2025

Reported by OpenAlex in 2 of 3 sources (CC0 flags, not verified indexing)DOAJSciELOOpenAlex Core

h-index

7

i10-index

4

From OpenAlex open data (CC0).

Editorial & policy12
Last DOAJ review date
2025-05-13
Added to DOAJ
2024-05-17
Declared plagiarism screening
Declared
Other fees beyond APC
No
Declared digital preservation
CLOCKSS
Observed digital preservation (Keepers registry)— snapshot 2026-10-05
CLOCKSS
Editorial board
Official page ↗
Open references deposited at Crossref
94%
ORCID iDs in current deposits
0%
Enrolled in Crossref Similarity Check
✓
Deposits licence metadata at Crossref
✓
APC waiver policy
View policy
Peer-review policy
View policy
Identity & continuity2
Source type
journal
NLM unique ID (PubMed)
9918645787006676
Classification2

Subjects (DOAJ)

Science: Biology (General)

LCC codes

QH301-705.5

Subject areas

  • Autophagy in Disease and Therapy
  • Autophagy in Disease and Therapy
  • Cell death mechanisms and regulation
  • Cell death mechanisms and regulation
  • Phagocytosis and Immune Regulation
  • Endoplasmic Reticulum Stress and Disease
  • Endoplasmic Reticulum Stress and Disease
  • Phagocytosis and Immune Regulation
Show all subjects (50)
  • Ferroptosis and cancer prognosis
  • Ferroptosis and cancer prognosis
  • MicroRNA in disease regulation
  • Immune Response and Inflammation
  • MicroRNA in disease regulation
  • Immune Response and Inflammation
  • CRISPR and Genetic Engineering
  • CRISPR and Genetic Engineering
  • Trace Elements in Health
  • Inflammasome and immune disorders
  • Mitochondrial Function and Pathology
  • Cancer, Lipids, and Metabolism
  • NF-κB Signaling Pathways
  • Ubiquitin and proteasome pathways
  • Cancer Cells and Metastasis
  • Trace Elements in Health
  • Cancer, Lipids, and Metabolism
  • Nuclear Structure and Function
  • NF-κB Signaling Pathways
  • RNA modifications and cancer
  • Inflammasome and immune disorders
  • Ubiquitin and proteasome pathways
  • Cellular transport and secretion
  • RNA Research and Splicing
  • Mitochondrial Function and Pathology
  • Cancer Cells and Metastasis
  • RNA modifications and cancer
  • Cancer Immunotherapy and Biomarkers
  • Sperm and Testicular Function
  • Acute Myeloid Leukemia Research
  • Malaria Research and Control
  • Computational Drug Discovery Methods
  • Environmental Toxicology and Ecotoxicology
  • Computational Drug Discovery Methods
  • Malaria Research and Control
  • Epigenetics and DNA Methylation
  • Neuroscience and Neuropharmacology Research
  • Cellular Mechanics and Interactions
  • Metal complexes synthesis and properties
  • Cancer Immunotherapy and Biomarkers
  • Sperm and Testicular Function
  • Metal complexes synthesis and properties

Keywords (DOAJ)

  • apoptosis
  • autophagy
  • ferroptosis
  • necroptosis
  • necrosis

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The journal's record in other sources:Web of Science MJLDOAJ

Everything shown here is produced automatically from named sources (DOAJ, OpenAlex, Crossref, PubMed, the Norwegian register) as of its capture date, with no human editing. Facts appear as recorded at the source; derived scores (PCI, PTD, PublishLens score) follow a published methodology. Not a judgement on the journal, not an official accreditation, not advice.

Wrong items are corrected automatically by re-reading the source (24 h for daily sources, 7 days for weekly ones) with a dated change log, and each item links to its source record. PublishLens-derived assessments are contested through the appeals mechanism (15 working days). Appeals · Indicator guide

Norwegian register data: Kanalregisteret (HK-dir, Norway) — CC BY 4.0 / NLOD — adapted: level extracted per year · OASPA member list — CC BY

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