About the journal
Contact is an open-access journal published by SAGE Publishing in United States, publishing in english. It is listed in DOAJ and PUBMED according to the sources read. Declared publication charge: 850 USD. Declared review time at DOAJ: 58 weeks. Facts are reproduced as recorded at their sources with their capture dates; PublishLens indicators are available after sign-in.
Basic facts
- Publisher
- SAGE Publishing
- ISSN
- 2515-2564
- Country
- United States
- Languages
- english
- Access
- Open access
- Declared publication charge
- 850 USD
- Articles in DOAJ
- 159
- Declared review time
- 58 weeks
- Declared peer-review model
- double_blind
- Official website
- https://journals.sagepub.com/home/ctc
Index listings
- DOAJ · 2022-04-21
- PUBMED · 2018-04-17
- Indexing
- DOAJ · PUBMED
- APC
- 850 USD
- Declared review time (DOAJ)
- 58 weeks — journal-declared only (not measured)
- Activity (OpenAlex)
- Active — latest works 2026
- PublishLens list status
- Shown after sign-in
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- Choose your university and rank below ↓
Methodology signals (dated facts from their sources)
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Descriptive metadata from public sources (DOAJ, OpenAlex, PubMed) as of the capture date shown — contains no assessment or judgment.
Articles in DOAJ
159
Most recent article
last month
Average review time (journal-declared via DOAJ)
58weeks
Works in OpenAlex
153
Last recorded publication year
2026
h-index
18
i10-index
48
From OpenAlex open data (CC0).
Editorial & policy11
- Last DOAJ review date
- 2024-04-04
- Added to DOAJ
- 2022-04-21
- Declared plagiarism screening
- Declared
- Other fees beyond APC
- No
- Observed digital preservation (Keepers registry)— snapshot 2026-10-05
- CLOCKSSPortico
- Editorial board
- Official page ↗
- Open references deposited at Crossref
- 96%
- ORCID iDs in current deposits
- 96%
- Enrolled in Crossref Similarity Check
- ✓
- Deposits licence metadata at Crossref
- ✓
- APC waiver policy
- View policy
- Peer-review policy
- View policy
Identity & continuity2
- Source type
- journal
- NLM unique ID (PubMed)
- 101729710
Classification4
Subjects (DOAJ)
LCC codes
Subject areas
- Cellular transport and secretion
- Cellular transport and secretion
- Endoplasmic Reticulum Stress and Disease
- Endoplasmic Reticulum Stress and Disease
- Lipid Membrane Structure and Behavior
- Lipid Membrane Structure and Behavior
- Mitochondrial Function and Pathology
- Mitochondrial Function and Pathology
Show all subjects (50)
- Lipid metabolism and biosynthesis
- Lipid metabolism and biosynthesis
- Photosynthetic Processes and Mechanisms
- Photosynthetic Processes and Mechanisms
- Autophagy in Disease and Therapy
- Autophagy in Disease and Therapy
- ATP Synthase and ATPases Research
- Calcium signaling and nucleotide metabolism
- ATP Synthase and ATPases Research
- Calcium signaling and nucleotide metabolism
- Adipose Tissue and Metabolism
- Adipose Tissue and Metabolism
- Peroxisome Proliferator-Activated Receptors
- Peroxisome Proliferator-Activated Receptors
- Biotin and Related Studies
- Ion Channels and Receptors
- Biotin and Related Studies
- Lysosomal Storage Disorders Research
- Ion Channels and Receptors
- Lysosomal Storage Disorders Research
- Fungal and yeast genetics research
- Fungal and yeast genetics research
- Ion channel regulation and function
- Metabolism and Genetic Disorders
- Metabolism and Genetic Disorders
- Ion channel regulation and function
- Erythrocyte Function and Pathophysiology
- Cholesterol and Lipid Metabolism
- Erythrocyte Function and Pathophysiology
- Cholesterol and Lipid Metabolism
- Genetic Neurodegenerative Diseases
- Genetic Neurodegenerative Diseases
- RNA Research and Splicing
- Glycosylation and Glycoproteins Research
- RNA and protein synthesis mechanisms
- RNA Research and Splicing
- Glycosylation and Glycoproteins Research
- Receptor Mechanisms and Signaling
- Sphingolipid Metabolism and Signaling
- RNA and protein synthesis mechanisms
- Sphingolipid Metabolism and Signaling
- Receptor Mechanisms and Signaling
Keywords (DOAJ)
- membrane contact sites
- intracellular contacts
- lipids
- membrane traffic
- cell signalling
- cellular homeostasis
Community confirmations — what users saw in MJL or Scopus Sources; not platform verification and never part of any score.
The journal's record in other sources:Web of Science MJLDOAJ